Intravenous Vitamin C

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** Doctors reported Monday cancer patients who were given large intravenous doses of Vitamin C over a period of several months had their lives extended and their tumors shrunk.
MAJOR BREAKTHROUGH REPORT
INTRAVENOUS VITAMIN C KILLS CANCER CELLS
Recall how hydrogen peroxide is poured on wounds to kill germs. Well now researchers clearly show high-dose vitamin C, when administered intravenously, can increase hydrogen peroxide (H2O2) levels within cancer cells and kills them. I.V. vitamin C was also demonstrated to kill germs and may be an effective therapy for infectious disease.
With a growing body of evidence mounting, National Institutes of Health (NIH) researchers conceded today that intravenous vitamin C may be an effective treatment for cancer. Last year the same researchers reported a similar study but the news media failed to publish it.
The latest study, published in the Proceedings of the National Academy of Sciences, confirms the work of Nobel-Prize winner Dr. Linus Pauling who conducted cancer research in the 1970s with vitamin C. Dr. Pauling's studies were discredited at the time by poorly conducted research studies at the Mayo Clinic.
Unlike cancer drugs, I.V. vitamin C selectively killed cancer cells, but not healthy cells, and showed no toxicity. The ability of intravenous vitamin C to kill lymphoma cells was remarkable – almost 100% at easily achievable blood serum concentrations.

4 replies

Please post a link to any articles on this new finding.

I find this very interesting. I know there were clinics that did the iv's and supposedly were shut down by the FDA. Apparently there are places where you can get the iv. One of the first books I read was The Cancer Battle Plan by Anne E Frahm. She was basically sent home from hospital to die. She was cured and it has been over 9 years. She had breast cancer ,but the point is she was so bad that her spine cracked from mestases. Interesting diet along with the vitamin C. I think anyone that wants it should be allowed to have it. Like anything there is a percentage where it may help and where it may do nothing. With this disease I believe you should have all the armour you want.

Valeriel,

I have been getting these same intravenous vitamin c treatments post surgery since July of last year. Started out every week, then every other week, then every three weeks and now only once a month. Plan to continue to do monthly treatments for a period of two years per my alternative doctor's recommendation.

Thanks for the info and God Bless,

Frank

Frank

I'm sorry not to have provided a link to my comment. I was not able to find one myself, however I found many interesting sources by googling Intravenous Vitamin C. Doctoryourself.com had protocols and how to get intravenous to hospitalized loved ones and this article:
How Vitamin C Stops Cancer

ScienceDaily (Sep. 12, 2007) — Nearly 30 years after Nobel laureate Linus Pauling famously and controversially suggested that vitamin C supplements can prevent cancer, a team of Johns Hopkins scientists have shown that in mice at least, vitamin C - and potentially other antioxidants - can indeed inhibit the growth of some tumors ¯ just not in the manner suggested by years of investigation.
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Health & Medicine


* Cancer

* Lung Cancer

* Brain Tumor

Plants & Animals


* Mice

* Biotechnology

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* Polyphenol antioxidant

* Vitamin E

* B vitamins

* Antioxidant

The conventional wisdom of how antioxidants such as vitamin C help prevent cancer growth is that they grab up volatile oxygen free radical molecules and prevent the damage they are known to do to our delicate DNA. The Hopkins study, led by Chi Dang, M.D., Ph.D., professor of medicine and oncology and Johns Hopkins Family Professor in Oncology Research, unexpectedly found that the antioxidants' actual role may be to destabilize a tumor's ability to grow under oxygen-starved conditions. Their work is detailed this week in Cancer Cell.

"The potential anticancer benefits of antioxidants have been the driving force for many clinical and preclinical studies," says Dang. "By uncovering the mechanism behind antioxidants, we are now better suited to maximize their therapeutic use."

"Once again, this work demonstrates the irreplaceable value of letting researchers follow their scientific noses wherever it leads them," Dang adds.

The authors do caution that while vitamin C is still essential for good health, this study is preliminary and people should not rush out and buy bulk supplies of antioxidants as a means of cancer prevention.

The Johns Hopkins investigators discovered the surprise antioxidant mechanism while looking at mice implanted with either human lymphoma (a blood cancer) or human liver cancer cells. Both of these cancers produce high levels of free radicals that can be suppressed by feeding the mice supplements of antioxidants, either vitamin C or N-acetylcysteine (NAC).

However, when the Hopkins team examined cancer cells from cancer-implanted mice not fed the antioxidants, they noticed the absence of any significant DNA damage. "Clearly, if DNA damage was not in play as a cause of the cancer, then whatever the antioxidants were doing to help was also not related to DNA damage," says Ping Gao, Ph.D, lead author of the paper.

That conclusion led Gao and Dang to suspect that some other mechanism was involved, such as a protein known to be dependent on free radicals called HIF-1 (hypoxia-induced factor), which was discovered over a decade ago by Hopkins researcher and co-author Gregg Semenza, M.D., Ph.D., director of the Program in Vascular Cell Engineering. Indeed, they found that while this protein was abundant in untreated cancer cells taken from the mice, it disappeared in vitamin C-treated cells taken from similar animals.

"When a cell lacks oxygen, HIF-1 helps it compensate," explains Dang. "HIF-1 helps an oxygen-starved cell convert sugar to energy without using oxygen and also initiates the construction of new blood vessels to bring in a fresh oxygen supply."

Some rapidly growing tumors consume enough energy to easily suck out the available oxygen in their vicinity, making HIF-1 absolutely critical for their continued survival. But HIF-1 can only operate if it has a supply of free radicals. Antioxidants remove these free radicals and stop HIF-1, and the tumor, in its tracks.

The authors confirmed the importance of this "hypoxia protein" by creating cancer cells with a genetic variant of HIF-1 that did not require free radicals to be stable. In these cells, antioxidants no longer had any cancer-fighting power.

The research was funded by the National Institutes of Health.

Authors on the paper are Dean Felsher of Stanford; and Gao, Huafeng Zhang, Ramani Dinavahi, Feng Li, Yan Xiang, Venu Raman, Zaver Bhujwalla, Linzhao Cheng, Jonathan Pevsner, Linda Lee, Gregg Semenza and Dang of Johns Hopkins.

Adapted from materials provided by Johns Hopkins Medical Institutions, via EurekAlert!, a service of AAAS.
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Johns Hopkins Medical Institutions (2007, September 12). How Vitamin C Stops Cancer. ScienceDaily. Retrieved February 22, 2008, from http://www.sciencedaily.com­ /releases/2007/09/070910132848.htm

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